Biology
12th Edition
ISBN: 9780134813448
Author: Audesirk, Teresa, Gerald, Byers, Bruce E.
Publisher: Pearson,
expand_more
expand_more
format_list_bulleted
Concept explainers
Videos
Textbook Question
Chapter 37.8, Problem 1TC
What might be the evolutionary advantage of allergic reactions? (Hint; Are there harmful substances or organisms that might provoke allergic reactions?)
Expert Solution & Answer
Want to see the full answer?
Check out a sample textbook solution
Students have asked these similar questions
Is their a correlation between age and allergic reaction?
Which of the followings is molecular pathology associated with ADA mutations that cause immunodeficiency?
a)
accumulation of deoxyadenosine and its precursors S-adenosylhomocysteine and deoxyadenosine triphosphate (dATP) with toxic effects on immature lymphocytes,
b)accumulation of deoxyguanosine and deoxyguanosine triphosphate, with toxic effects on immature lymphocytes
From: "Towards a universal flu vaccine"
Each
year,
the flu vaccine includes antigens from two strains of Influenza A and two
strains of Influenza B. These antigens are from the head of the H spikes an area that
sticks out from the virus and so, is very easy for human immune cells to detect.
However, the problem with using the head of the H spike as an antigen is...
O The H spike head mutates very rapidly, so quickly changes to forms not recognized by human
immune cells.
O The H spike head is exposed on the surface of the virus for only a short period of time.
Viruses quickly pull the H spikes back inside the virus, shiclding them from human immune
cells.
O The H spike head is often too big for the human immune cells to attack.
O The H spike head is often too small for the human immune cells to bind to.
Chapter 37 Solutions
Biology
Ch. 37.1 - compare and contrast the terms microbe and...Ch. 37.1 - Prob. 2CYLCh. 37.2 - Prob. 1TCCh. 37.2 - If phagocytes kill most species of bacteria that...Ch. 37.2 - describe the external barriers to Infection,...Ch. 37.2 - Prob. 2CYLCh. 37.3 - Prob. 1CYLCh. 37.3 - Prob. 2CYLCh. 37.4 - Prob. 1TCCh. 37.4 - Prob. 1CYL
Ch. 37.4 - Prob. 2CYLCh. 37.4 - Prob. 3CYLCh. 37.5 - Prob. 1CYLCh. 37.5 - Prob. 2CYLCh. 37.6 - Prob. 1CYLCh. 37.6 - Prob. 2CYLCh. 37.7 - Prob. 1HYEWCh. 37.7 - Prob. 1TCCh. 37.7 - explain how antibiotics, antiviral drugs, and...Ch. 37.7 - Prob. 2CYLCh. 37.8 - What might be the evolutionary advantage of...Ch. 37.8 - Prob. 1CYLCh. 37.9 - Prob. 1CYLCh. 37.9 - describe current medical treatments against...Ch. 37.9 - Prob. 1CTCh. 37 - Prob. 1MCCh. 37 - Prob. 2MCCh. 37 - Molecules that label your cells as self are a....Ch. 37 - Prob. 4MCCh. 37 - The immune system usually does not attack your...Ch. 37 - External defenses against microbial invasion...Ch. 37 - Prob. 2FIBCh. 37 - Prob. 3FIBCh. 37 - Prob. 4FIBCh. 37 - Prob. 5FIBCh. 37 - Prob. 6FIBCh. 37 - Prob. 1RQCh. 37 - Prob. 2RQCh. 37 - Describe humoral immunity and cell-mediated...Ch. 37 - Prob. 4RQCh. 37 - Prob. 5RQCh. 37 - Prob. 6RQCh. 37 - Prob. 7RQCh. 37 - What are memory cells? How do they contribute to...Ch. 37 - Prob. 9RQCh. 37 - Prob. 10RQCh. 37 - Prob. 11RQCh. 37 - Prob. 12RQCh. 37 - Prob. 13RQCh. 37 - Why is it essential that antibodies and T-cell...Ch. 37 - Prob. 2AC
Knowledge Booster
Learn more about
Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- Describe in detail the effect of loss or mutation of LAT & Blk of the following molecules/proteins/genes/domains in immune signaling. Be sure to include the normal function and the function effected by the mutation. Include all relevant players and their roles. You may use diagrams if needed.arrow_forwardExplain why transplanting skin graft from the black mouse to white mouse results in rejection (A) Why (A) is it called a primary response?arrow_forwardWhich of the following is not a symptom of an allergic reaction? Group of answer choices Asthma or wheezing Development of hives or rashes Irritated or reddened eyes Tingling sensation in the mouth and tongue Sudden increase in blood pressurearrow_forward
- For the following mechanism that is thought to contribute to maternal–fetal tolerance, state whether you would consider it to be an example of T cell tolerization, control of responses by regulatory lymphocytes, immune deviation or immune privilege: a) Tissues at the maternal–fetal interface are populated with non-professional APCs that lack costimulatory moleculesarrow_forwardHuman immune system a) Pathogen-associated molecular patterns are conserved molecular structures produced by microorganisms, but not by host cells. b) A virus-infected host cell that displays a viral antigen via MHC class I molecules may become a target of cytotoxic T cells. c) Secreted MHC Class I proteins present in blood plasma can function as opsonins. d) Both (a) and (b) are correct and (c) is incorrect e) Statements (a), (b) and (c) are all correctarrow_forwardMake an illustration that connects the events in inflammation mediated by chemical substances. Use the following words to come up with the output connected by arrows. 1. Modified proteins 16. C142356789 2. Kininogen 3. Prostaglandins 4. Clotting system 5. Antigen 17. Kallikrein 18. Platelets 19. Serotonin 20. Kinins 21. C1423 22. Anaphylotoxins 6. Lysosomal enzymes 7. Antibody 8. C1 9. Kallikreinogen 23. Chemotactic factor 24. Serum factors 10. C142 25. C14235 11. Antigen/Antibody 12. Cells migration 26. C1423567 27. Injurious stimuli 13. Histamine 28. Acute inflammation 14. Plasmin system 29. Increased vascular permeability 15. C3 30. Chronic inflammationarrow_forward
- You are in the process of developing a monoclonal antibody against an Influenza virus spike protein "H7." You have isolated the B cells producing antibodies against the viral H7 protein and fused those specific B cells with a unique kind of mutant myeloma cells. The myeloma cells can't make any of the three following enzymes; dihydrofolate reductase (DHFR), thymidine kinase (TK), and hypoxanthine-guanosine phosphoribosyl transferase (HGPRT). The mutant unfused myeloma cells are grown in the laboratory in a nucleotide supplemented medium. For selecting the hybridomas, you have used a modified medium HT that does not contain any aminopterin but contains hypoxanthine and thymidine. Which of the following result do you expect to see from your experiment, and what problem you may encounter in the future after the hybridoma selection?arrow_forwardplease answer # 1a and 1 1a) LIST and DESCRIBE at least 5 Molecules and Cell types that are shared and interconnect the INNATE Defenses of the body with the ACQUIRED / Adaptive Immunity Include both proteins and signaling ligands. DEFINE the defensive functions of EACH shared component and Use simple DRAWINGS to illustrate the interactions. EXPLAIN HOW they connect the immune and innate pathways.arrow_forwardExplain the multifaceted role of interferons as first responders to viral infection: What types of interferons are activated by viruses at the earliest stages of infection? How are interferons activated? What types of stimuli activate interferons at the earliest stages of viral infection.? Identify the receptors to which interferon binds and how their activation affects gene expression?arrow_forward
- Cyclosporin A and rapamycin are each used as T cell immunosuppressants. They share the property of binding to immunophilin molecules in T cells as the initial step in their mechanisms of action. However, in the case of cyclosporin A, the drug:immunophilin complex binds to and inhibits the protein phosphatase calcineurin, whereas the rapamycin:immunophilin complex binds to and inhibitors mTOR. As a consequence, Cyclosporin A, but not rapamycin, blocks cytokine production by T cells. Both cyclosporin A and rapamycin block cytokine production by T cells. Rapamycin, but not cyclosporin A, blocks T cell proliferation. Neither rapamycin nor cyclosporin A block T cell proliferation. Both cyclosporin A and rapamycin inhibit co-stimulatory signaling through CD28 on T cells.arrow_forwardA young child with cretinism is not growing and her parents would like her to take synthetic hGH. Will this treatment help the child? Explain your answerarrow_forwardTo avoid the potential for activating self-reactive T cells that might cause autoimmunity, naive T cell activation has stringent requirements for co-stimulatory signals in addition to the engagement of T-cell receptors with peptide: MHC complexes. Yet this requirement is abandoned once T cells have differentiated into effector cells. Name two effector T cell functions that would fail if effector T cells also required T-cell receptor signals plus co-stimulatory signals through CD28 to elicit their effector responses.arrow_forward
arrow_back_ios
SEE MORE QUESTIONS
arrow_forward_ios
Recommended textbooks for you
Human Anatomy & Physiology (11th Edition)BiologyISBN:9780134580999Author:Elaine N. Marieb, Katja N. HoehnPublisher:PEARSON
Biology 2eBiologyISBN:9781947172517Author:Matthew Douglas, Jung Choi, Mary Ann ClarkPublisher:OpenStax
Anatomy & PhysiologyBiologyISBN:9781259398629Author:McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa StouterPublisher:Mcgraw Hill Education,
Molecular Biology of the Cell (Sixth Edition)BiologyISBN:9780815344322Author:Bruce Alberts, Alexander D. Johnson, Julian Lewis, David Morgan, Martin Raff, Keith Roberts, Peter WalterPublisher:W. W. Norton & Company
Laboratory Manual For Human Anatomy & PhysiologyBiologyISBN:9781260159363Author:Martin, Terry R., Prentice-craver, CynthiaPublisher:McGraw-Hill Publishing Co.
Inquiry Into Life (16th Edition)BiologyISBN:9781260231700Author:Sylvia S. Mader, Michael WindelspechtPublisher:McGraw Hill Education
Human Anatomy & Physiology (11th Edition)
Biology
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:PEARSON
Biology 2e
Biology
ISBN:9781947172517
Author:Matthew Douglas, Jung Choi, Mary Ann Clark
Publisher:OpenStax
Anatomy & Physiology
Biology
ISBN:9781259398629
Author:McKinley, Michael P., O'loughlin, Valerie Dean, Bidle, Theresa Stouter
Publisher:Mcgraw Hill Education,
Molecular Biology of the Cell (Sixth Edition)
Biology
ISBN:9780815344322
Author:Bruce Alberts, Alexander D. Johnson, Julian Lewis, David Morgan, Martin Raff, Keith Roberts, Peter Walter
Publisher:W. W. Norton & Company
Laboratory Manual For Human Anatomy & Physiology
Biology
ISBN:9781260159363
Author:Martin, Terry R., Prentice-craver, Cynthia
Publisher:McGraw-Hill Publishing Co.
Inquiry Into Life (16th Edition)
Biology
ISBN:9781260231700
Author:Sylvia S. Mader, Michael Windelspecht
Publisher:McGraw Hill Education
Immune System Diseases and Disorders; Author: Heather Davis;https://www.youtube.com/watch?v=3lIkxNv7MVI;License: Standard youtube license