Fill out the table below. Determine whether the rate of the metabolic pathways will increase or decrease in the presence of a hormone. Insulin Glucagon Glycolysis Hexose monophosphate pathway Gluconeogenesis Glycogenolysis
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- Discuss the synthesis and breakdown of glycogen and how the processes are regulated in response xto hormone action. also describe the various types of glycogen storage diseasesPeople with non-insulin-dependent diabetes mellitus are generally not ketosis prone. This is thought to be a result of: the lack of increase in glucagons in the se individuals the presence of insulin in the individuals their obesity the fact that, their blood glucose levels do not tend to rise significantly Metabolic actions of insulin include all of the following except: increased glycogenesis decreased gluconeogenesis increased basal metabolic rate increased skeletal muscle amino acid up-take all of the above are metabolic actions of insulinBlood sugar homeostasis: Indicate the order of events that will happen following blood glucose increase after a meal. Increased blood glucose 1 2 3 4 LO 5 6 :::: Glucose transport via GLUT4 Glucose transport into pancreatic cells Insulin binds to insulin receptor Insulin release from pancreatic beta-cells Second messenger signaling pathway is activated GLUT4 moves to plasma membrane Decreased blood glucose AND muscle makes ATP and glycogen
- This is a question assigned after reading the case study available here https://sciencecases.lib.buffalo.edu/files/statins.pdf HMG CoA reductase is activated by insulin signaling, and inhibited by glucagon signaling. Explain why insulin resistance and type II diabetes often are accompanied by elevated cholesterol levels.Briefly describe the insulin signalling pathway. Explain what insulin resistance is and how it is linked to obesity.Complete the following chart about PDHK activity by determining if the molecule would be in high or low concentration or which pathway would be active. Molecule or pathway Low Energy State High Energy State Руruvate ATP/ADP Acetyl-CoA/CoA NADH/ NAD* Glycolysis/gluconeogenesis Insulin/Glucagon
- . Explain why insulin can stimulate the activation PKB of but not the activation of PK.Choose the correct. The major action of insulin is: A. Conversion of glucose to glycogenB. ProteolysisC. Conversion of fatty acids to glucoseD. GlycogenolysisE. GluconeogenesisWhich of these enzymes/complexes will insulin activate in the liver? Choose all that apply. Glycogen synthase Glycogen phosphorylase Acetyl-CoA carboxylase Carnitine shuttle PFK-1 FBPase-1
- Compare and contrast INSULIN and GLUCAGON using a Venn diagram.Insulin stimulated glucose uptake takes place in all of the following except- 1. heart 2. skeletal muscle 3. liver 4. adipose tissueLow insulin levels and low plasma amino acid levels Protein Catabolism Olher amino acids alpha-KG Glutamate Giutamne Gutamine Glutamate SO H Cystie)ine EGSH Cystfe)ineCyst(eline- Urea Skeletal Muscle Liver To the best of your abilities, explain the image above. Do not include other "unnecessary" things that you cannot see in the picture.
