Biology
5th Edition
ISBN: 9781260487947
Author: BROOKER
Publisher: MCG
expand_more
expand_more
format_list_bulleted
Concept explainers
Videos
Textbook Question
Chapter 42.4, Problem 1EQ
What key prior observation led Loewi to develop his hypothesis of how nerves stimulate or inhibit heart muscle contractions?
Expert Solution & Answer
Want to see the full answer?
Check out a sample textbook solution
Students have asked these similar questions
The tetanus toxin tetanospasmin blocks release of the neurotransmitter (GABA) that inhibits motor neurons and decreases muscle contraction. This causes persistence of the signal to contract and the resulting violent muscle contraction known as tetanic spasm. Compare the sustained contraction of muscle cells in rigor mortis with muscle contraction caused by the action of tetanospasmin on motor neurons.
Describe experiments using marker molecules that have been done to elucidate important properties of (1) tight junctions and (2) gap junctions, and propose one additional experiment you could do with each junction to gain a greater understanding of that junction's properties.
The experiment that crossed the nerve from a muscle that had a short contraction time "a fast muscle" to a muscle with a slower contraction profile and from the slower muscle to the faster muscle demonstrated that the nerve or activity of the muscle had no effect on properties of the muscle fibers.
a.) True
b.) False
How does Piaget's theory play out in a preschool setting?
Chapter 42 Solutions
Biology
Ch. 42.1 - Prob. 1CCCh. 42.2 - Prob. 1CSCh. 42.3 - Prob. 1CSCh. 42.3 - Prob. 2CSCh. 42.3 - Prob. 1CCCh. 42.3 - Prob. 2CCCh. 42.4 - What key prior observation led Loewi to develop...Ch. 42.4 - CoreSKILL The results of Loewis experiment...Ch. 42.4 - Prob. 3EQCh. 42 - Prob. 1TY
Ch. 42 - Prob. 2TYCh. 42 - The myelin sheath a. is produced by neurons in the...Ch. 42 - Prob. 4TYCh. 42 - Prob. 5TYCh. 42 - Which of the following contribute(s) to the...Ch. 42 - Prob. 7TYCh. 42 - Prob. 8TYCh. 42 - Prob. 9TYCh. 42 - Prob. 10TYCh. 42 - Prob. 1CQCh. 42 - Prob. 2CQCh. 42 - Prob. 3CQCh. 42 - Describe the difference between an electrical...Ch. 42 - Prob. 2COQ
Additional Science Textbook Solutions
Find more solutions based on key concepts
CAUTION How can evolutionary fitness be estimated? a. Document how long individuals survive. b. Count the numbe...
Biological Science (6th Edition)
Explain why hyperthermophiles do not cause disease in humans.
Microbiology with Diseases by Taxonomy (5th Edition)
Some people consider Pasteur or Koch to be the Father of Microbiology, rather than Leeuwenhoek. Why might they ...
Microbiology with Diseases by Body System (4th Edition)
1. Genetics affects many aspects of our lives. Identify three ways genetics affects your life or the life of a ...
Genetic Analysis: An Integrated Approach (2nd Edition)
6. How can you use the features found in each chapter?
Human Anatomy & Physiology (2nd Edition)
Describe Mendels conclusions about how traits are passed from generation to generation.
Concepts of Genetics (11th Edition)
Knowledge Booster
Learn more about
Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- Please answer 1 and 2. 1) Calcium enters the presynaptic neuron by: a) simple diffusion. b) facilitated diffusion. c) active transport. 2) Neurotransmitters directly affect: a)voltage-gated channels. b) ligand-gated channels. c) mechanosensitive channels.arrow_forwardWhen the neurotransmitter acetylcholine (ACh) binds the acetylcholine receptor (a GPCR) on muscle cells, it causes them to contract. ZIGGY, a chemical analog of ACh, also binds to the same acetylcholine receptor on muscle cells, but instead causes the muscle cells to relax. For this reason, it is sometimes prescribed as a muscle relaxer. Explain in 3-4 sentences how ZIGGY could cause muscle relaxation. How can both ZIGGY and ACh bind the same GPCR? And then how can they have different effects on the cells, despite binding to the same receptor on the same cells?arrow_forward"Motor neurons trigger action potentials in muscle cell membranes that open voltage-sensitive Ca2+ channels in T tubules, allowing extracellular Ca2+ to enter the cytosol, bind to troponin C, and initiate rapid muscle contraction", is true or false.arrow_forward
- A researcher is investigating the effects of a toxin, isolated from the skin of a South American tree frog. The researcher is exploring the effect of that toxin on synaptic transmission at the mammalian skeletal neuromuscular junction. Their previous studies established that the toxin had no direct effect upon skeletal muscle contractility, but it did block muscle contractility elicited indirectly via electrical stimulation of the motor nerve. To establish how the toxin influenced synaptic transmission, in vitro studies were performed using the mouse phrenic nerve hemidiaphragm skeletal muscle preparation, bathed in a buffered saline solution containing a raised magnesium and lowered calcium concentration. Under these conditions intracellular recording determined the effect of the toxin on the amplitude (in mV) of miniature endplate potentials (mepps) and endplate potentials (epps) – see Table 1. Mean mepp amplitude Mean epp amplitude Control 0.5 mV 8 mV Toxin 0.5 mV 2 mV Table 1: Mean…arrow_forwardIn describing a positive feedback loop, which of the following would be true? a) the sensor and control center are always the same b) the effector signals the control center to shut off the initial stimulus c) the effect shuts off the initial stimulus d) the effect amplifies the initial stimuarrow_forwardHow does pair pulse-facilitation demonstrate Hebb's postulate and why is it transient? (please explain very simply)arrow_forward
- Many neurotoxins have been used extensively in physiological studies of neurons. When comparing tetrodotoxin found in puffer fish and batrachotoxins from poison dart-frogs, the former had no effect on a neurons resting potential but completely stopped its action potential, whereas the later toxin immediately initiates depolarization of a neuron but prevents repolarization. Can your group hypothesize how these toxins affect the nerve transmission differently?arrow_forwardNeurons release norepinephrine or acetylcholine onto their target organ. O It regulates heart rate Question 30 While walking barefoot along the beach, you step on a shard of glass and reflexively lift your injured foot. Which path did the reflex to lift your foot take? O Motor nerve → spinal cord → brain → spinal cord → sensory nerve → skeletal muscle O Sensory nerve → spinal cord → brain → motor nerve → skeletal muscle O Sensory nerve → spinal cord → motor nerve → skeletal muscle O Sensory nerve → motor nerve → spinal cord → brain Question 31 Generally, nerve impulses do not move "backward" along a series of neurons. What is the reason for this? 1 pts 80 1 ptarrow_forwardPlease explain the mechanismarrow_forward
arrow_back_ios
SEE MORE QUESTIONS
arrow_forward_ios
Recommended textbooks for you
Human Heredity: Principles and Issues (MindTap Co...BiologyISBN:9781305251052Author:Michael CummingsPublisher:Cengage Learning
Human Heredity: Principles and Issues (MindTap Co...
Biology
ISBN:9781305251052
Author:Michael Cummings
Publisher:Cengage Learning
GCSE PE - ANTAGONISTIC MUSCLE ACTION - Anatomy and Physiology (Skeletal and Muscular System - 1.5); Author: igpe_complete;https://www.youtube.com/watch?v=6hm_9jQRoO4;License: Standard Youtube License