The rising phase of the action potential is due to:Na+ influx through voltage-gated Na+ channels.Na+ efflux through voltage-gated Na+ channels.K+ influx through voltage-gated K+ channels.K* efflux through voltage-gated K+ channels.O Na+ influx through Na+ leak channels.Na efflux through Na+ leak channels.K+ influx through K+ leak channels.O K+ efflux through K+ leak channels.

Introduction An electrical signal called an action potential passes briefly over the membrane of a…

The rising phase of the action potential is due to: O Na+ influx through voltage-gated Na+ channels. O Na+ efflux through voltage-gated Na+ channels. OK* influx through voltage-gated K+ channels. OK* efflux through voltage-gated K+ channels. O Nat influx through Nat leak channels. O Nat efflux through Nat leak channels. OK* influx through K+ leak channels. K+ efflux through K+ leak channels.

The rising phase of the action potential is due to: O Na+ influx through voltage-gated Na+ channels. O Na+ efflux through voltage-gated Na+ channels. OK* influx through voltage-gated K+ channels. OK* efflux through voltage-gated K+ channels. O Nat influx through Nat leak channels. O Nat efflux through Nat leak channels. OK* influx through K+ leak channels. K+ efflux through K+ leak channels.

Human Physiology: From Cells to Systems (MindTap Course List)

9th Edition

ISBN:9781285866932

Author:Lauralee Sherwood

Publisher:Lauralee Sherwood

Chapter4: Principles Of Neural And Hormonal Communication

Section: Chapter Questions

Problem 17RE

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Draw and label an action potential, indicating the ion movements responsible for the rising phase and the falling phase.
The falling phase of the action potential is due to: Na+ influx through voltage-gated Na+ channels. O Na* efflux through voltage-gated Na* channels. OK* influx through voltage-gated K+ channels. O K+ efflux through voltage-gated K channels. O Nat influx through Na* leak channels. Na efflux through Na+ leak channels. O K+ influx through K+ leak channels. O K+ efflux through K+ leak channels.
Some have compared the "all or none" action potential to flushing a toilet. The relative refractory period (when the water level in the tank is below what it is at rest ) is set by: the opening of the voltage gated sodium channels O the inactivation of voltage gated potassium channels the opening of voltage gated potassium channels the inactivation of voltage gated sodium channels
Which of the following does not influence (i.e has no impact on) the value of the Nernst potential for Na+? The extracellular Na+ concentration The intracellular Na+ concentration The charge on Na+ The number of Na+ channels open
During the action potential waveform, the voltage returns to the resting membrane potential at -70mV through the: closing of potassium channels. opening of chloride channels. O opening of sodium channels. opening of potassium channels.
The traces below show the action potential waveforms in an external meidum of different concentrations of * 40 100% 50% 33% 40 1 2 time (msec) calcium chloride potassium O sodium
Describe the action potential in terms of the different functional states of the voltage- gated Na+ membrane channels (Note: there are three states)
Dendrotoxins, produced by the mamba snakes (Dendroaspis), are inhibitors of the voltage-gated K+ channels. What phase of the action potential would this toxin affect? How would it affect ion permeability during this phase? How would ion movement be affected?
Some have compared the "all or none" action potential to flushing a toilet. The absolute refractory period (when no amount of pressing the lever will produce another flush) is set by: the inactivation of voltage gated potassium channels the inactivation of voltage gated sodium channels the opening of voltage gated sodium channels the inactivation of voltage gated chloride channels
Epilepsy is a condition which results in seizures stemming from excessive or abnormal activity of neurons. This can occur either from hyperexcitability of excitatory neurons, or impairment of inhibitory neurons. That is to say, either the excitatory pathways become overactive, or the inhibitory pathways, designed to temper the excitatory pathways, are not active enough. Much of the research done on epilepsy focuses on voltage-gated sodium channels, and to date over 700 different mutations to the channel have been identified as playing a role in epilepsy. The means by which these mutations contribute to epilepsy is quite complex, but for the sake of this CAL, let's simplify and apply what we have learned so far to identify potential mechanisms for this condition. In what way could voltage-gated sodium channels be affected in excitatory neurons which would increase the likelihood of the neuron firing an action potential? (one correct answer) The inactivation gate is slower to close. The…
Once an action potential reaches a neurotransmitter release site, the release of the neurotransmitter is triggered by: The influx of Na* through voltage-gated channels. O The efflux of Na* through voltage-gated channels. The influx of K* through voltage-gated channels. The efflux of K+ through voltage-gated channels. The influx of Ca²+ through voltage-gated channels. O The efflux of Ca2+ through voltage-gated channels.
dendrotoxins, produced by the mamba snakes are inhibitors of the voltage gated k+ channel. what phase of the action potential with this toxin effect? how would it affect ion permeability during this phase howard ion movement be affected?