Describe how chymotrypsin is activated via a cascade mechanism in the gut. How does it incorporate a positive feedback loop? How does it protect against acute pancreatitis?
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Describe how chymotrypsin is activated via a cascade mechanism in the gut.
How does it incorporate a positive feedback loop?
How does it protect against acute pancreatitis?
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- Orlistat is a weight loss drug that inhibits pancreatic lipase. Which of the following effects would you NOT expect to see in patients taking this drug? O a) Increased amounts of triglycerides in the faeces O b) Decreased concentration of chylomicrons in the lymphatic system O c) Increased concentration of fatty acids in the lumen of the small intestine O d) A decreased concentration of VLDL in the bloodLeptin inhibit eating by: a) inhibiting AgRP/NPY neurons b) activating melanocortin neurons c) Both a and b d) Neither a nor bHow does gastrointestinal tract evade dehydration? 1) Alternating wave-like contraction and relaxatio of the oral (mouth) muscles reabsorbs water which will be transported to the urinary system 2) water is an important digestive enzyme in the stomach. 3) Normal peristaltic movements in the large intestine ensure reabsorption of water that will be delivered to the circulatory system. 4) the water we drink will be mostly absord by the blood vessels attached to the esophagus and stomach. Skip question
- The small intestine secretes CCK to a) assist in the breakdown of fats b) stimulate release of pancreatin c) increase bile storage d) inhibit gastrin synthesis through negative feedback e) counteract the presence of secretinNonsteroidal anti-inflammatory drugs inhibit the synthesis of prostaglandins, and can produce peptic ulcers. What function is normally ascribed to endogenous prostaglandins in the gut? Why prostaglandin synthesis inhibition can lead to the development of gastric ulcers? What preventive measures can be used?How are the protein-digesting enzymes in pancreatic juice activated? What is the logic of this arrangement?
- How does pepsinogen change into its active form?Under what conditions does the active functioning of pancreatic lipase occur? What is its mechanism of action?In upper gastrointestinal bleeding, without knowing the cause or source of bleeding, why do we give proton pump inhibitors (PPIS, e.g. omeprazole)? What is the role of these, if the source of bleeding is not peptic or duodenal ulcer?
- Zollinger-Ellison syndrome is a peptic ulcer disease characterized by overproduction of gastric acid. Which two types of stomach cells are most likely involved in Zollinger-Ellison Syndrome?which substance bring excess lipoprotein in the bloodstream back to the liver? a) high density lipoproteins b)low density lipoproteins c)very low density lipoproteins both b and c all of the aboveA patient came to a dental clinic with complaints of dry mouth and multiple dental caries lesions. In the course of collecting anamnesis, it was found that an allergic patient takes antihistamines that block the H2 histamine receptors of smooth muscle cells of the salivary glands. It is known that histamine is a mediator that stimulates the secretion of saliva. Explain the mechanism of action of these drugs if it is known that the next day after discontinuation of the drug, the patient's dryness disappeared. For this: а) write the classification of inhibitors according to the mechanism of action b) indicate what type of inhibitors the drugs are taken by the patient; confirm your conclusion in writing c) guess what structure this drug may have d) give a picture illustrating the action of this type of inhibitors.
